Description

β Amyloid(28-35) human is a β-amyloid peptide (Abeta), a lipid-induced amyloid core fragment. β Amyloid oligomers are neurotoxic, and β Amyloid(28-35) human can interact with neuronal membranes, regulate secondary structure and neurotoxicity, and cause Alzheimer’s disease. β Amyloid(28-35) human has anisotropic effects on the acidic phospholipid DPH, resulting in enhanced internal fluidity of lipid membrane bilayers[1].—–C36H67N9O9S—-[1]Nagarajan S, et al. Lipid-induced conformational transition of the amyloid core fragment Abeta(28-35) and its A30G and A30I mutants. FEBS J. 2008 May;275(10):2415-27.–182945-56-8–802.04—-O=C(NCC(N[C@@H](C)C(N[C@@H]([C@@H](C)CC)C(N[C@@H]([C@@H](C)CC)C(NCC(N[C@@H](CC(C)C)C(N[C@@H](CCSC)C(O)=O)=O)=O)=O)=O)=O)=O)[C@H](CCCCN)N–Neurological Disease–10 mM in DMSO–Amyloid-β—-Neuronal Signaling–Peptides