Description

Nitrosoglutathione (GSNO), a exogenous NO donor and a substrate for rat alcohol dehydrogenase class III isoenzyme, inhibits cerebrovascular angiotensin II-dependent and -independent AT1 receptor responses[1][2][3][4].–20°C, 3 years (Powder)-Applications–Formula-C10H16N4O7S-Citation–References-[1]Marie-Lynda Bouressam, et al. S-nitrosoglutathione inhibits cerebrovascular angiotensin II-dependent and -independent AT 1 receptor responses: A possible role of S-nitrosation. Br J Pharmacol. 2019 Jun;176(12):2049-2062.|[2]D E Jensen, et al. S-Nitrosoglutathione is a substrate for rat alcohol dehydrogenase class III isoenzyme. Biochem J. 1998 Apr 15;331 ( Pt 2)(Pt 2):659-68.|[3]Caneta Brown, et al. The effects of S-nitrosoglutathione and S-nitroso-N-acetyl-D, L-penicillamine in a rat model of pre-eclampsia. J Nat Sci Biol Med. 2013 Jul;4(2):330-5.|[4]Yur-Ren Kuo, et al. Nitrosoglutathione promotes flap survival via suppression of reperfusion injury-induced superoxide and inducible nitric oxide synthase induction. J Trauma. 2004 Nov;57(5):1025-31.|[5]Eva Nozik-Grayck, et al. Pulmonary vasoconstriction by serotonin is inhibited by S-nitrosoglutathione. Am J Physiol Lung Cell Mol Physiol. 2002 May;282(5):L1057-65.-CASNumber-57564-91-7-MolecularWeight-336.32-Compound Purity-90.56-SMILES-OC(CNC([C@H](CSN=O)NC(CC[C@H](N)C(O)=O)=O)=O)=O-Research_Area-Inflammation/Immunology; Cardiovascular Disease-Solubility-H2O : 16.67 mg/mL (ultrasonic;warming;heat to 60°C)-Target-Angiotensin Receptor;Endogenous Metabolite-Isoform-AT1 Receptor;Human Endogenous Metabolite-Pathway-GPCR/G Protein;Metabolic Enzyme/Protease-MCE Product type-Dye Reagents